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mouse anti n icd  (Developmental Studies Hybridoma Bank)


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    Developmental Studies Hybridoma Bank mouse anti n icd
    Notch is required for premature activation of Notch target expression and Cut downregulation in CAF1-p105/p180-deficient follicle cells at the mitotic stage. (A–A‴) Knockdown of Notch restored Cut expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in A) at the mitotic stage. Cut staining is shown in A′. (B–B‴) Knockdown of Notch abolished the induction of E(spl)mβ-CD2 expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in B) at the mitotic stage. CD2 staining is shown in B′. (C–C‴) Knockdown of Notch restored Eya expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in C) at the mitotic stage. Eya staining is shown in C′. (D–D‴) Knockdown of Notch restored Cut expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in D) at the mitotic stage. Cut staining is shown in D′. (E–E‴) Knockdown of Notch abolished the induction of Hnt expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in E) at the mitotic stage. Hnt staining is shown in (E′). (F–F‴) Immunostaining of the Notch <t>intracellular</t> <t>domain</t> (NICD) was performed to examine the knockdown efficiency of the Notch RNAi in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in F). NICD staining is shown in F′. DNA staining with DAPI is shown in A‴,B‴,C‴,D‴,E‴,F‴. The genotypes of GFP-negative and GFP-positive cell clones are indicated in black and green boxes, respectively, on the left sides of image panels. Scale bars: 10 μm.
    Mouse Anti N Icd, supplied by Developmental Studies Hybridoma Bank, used in various techniques. Bioz Stars score: 95/100, based on 57 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+n+icd/pmc06362395-336-18-27?v=Developmental+Studies+Hybridoma+Bank
    Average 95 stars, based on 57 article reviews
    mouse anti n icd - by Bioz Stars, 2026-08
    95/100 stars

    Images

    1) Product Images from "Inhibition of Notch signaling by the p105 and p180 subunits of Drosophila chromatin assembly factor 1 is required for follicle cell proliferation"

    Article Title: Inhibition of Notch signaling by the p105 and p180 subunits of Drosophila chromatin assembly factor 1 is required for follicle cell proliferation

    Journal: Journal of Cell Science

    doi: 10.1242/jcs.224170

    Notch is required for premature activation of Notch target expression and Cut downregulation in CAF1-p105/p180-deficient follicle cells at the mitotic stage. (A–A‴) Knockdown of Notch restored Cut expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in A) at the mitotic stage. Cut staining is shown in A′. (B–B‴) Knockdown of Notch abolished the induction of E(spl)mβ-CD2 expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in B) at the mitotic stage. CD2 staining is shown in B′. (C–C‴) Knockdown of Notch restored Eya expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in C) at the mitotic stage. Eya staining is shown in C′. (D–D‴) Knockdown of Notch restored Cut expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in D) at the mitotic stage. Cut staining is shown in D′. (E–E‴) Knockdown of Notch abolished the induction of Hnt expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in E) at the mitotic stage. Hnt staining is shown in (E′). (F–F‴) Immunostaining of the Notch intracellular domain (NICD) was performed to examine the knockdown efficiency of the Notch RNAi in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in F). NICD staining is shown in F′. DNA staining with DAPI is shown in A‴,B‴,C‴,D‴,E‴,F‴. The genotypes of GFP-negative and GFP-positive cell clones are indicated in black and green boxes, respectively, on the left sides of image panels. Scale bars: 10 μm.
    Figure Legend Snippet: Notch is required for premature activation of Notch target expression and Cut downregulation in CAF1-p105/p180-deficient follicle cells at the mitotic stage. (A–A‴) Knockdown of Notch restored Cut expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in A) at the mitotic stage. Cut staining is shown in A′. (B–B‴) Knockdown of Notch abolished the induction of E(spl)mβ-CD2 expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in B) at the mitotic stage. CD2 staining is shown in B′. (C–C‴) Knockdown of Notch restored Eya expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in C) at the mitotic stage. Eya staining is shown in C′. (D–D‴) Knockdown of Notch restored Cut expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in D) at the mitotic stage. Cut staining is shown in D′. (E–E‴) Knockdown of Notch abolished the induction of Hnt expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in E) at the mitotic stage. Hnt staining is shown in (E′). (F–F‴) Immunostaining of the Notch intracellular domain (NICD) was performed to examine the knockdown efficiency of the Notch RNAi in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in F). NICD staining is shown in F′. DNA staining with DAPI is shown in A‴,B‴,C‴,D‴,E‴,F‴. The genotypes of GFP-negative and GFP-positive cell clones are indicated in black and green boxes, respectively, on the left sides of image panels. Scale bars: 10 μm.

    Techniques Used: Activation Assay, Expressing, Knockdown, Clone Assay, Staining, Mutagenesis, Immunostaining



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    Notch is required for premature activation of Notch target expression and Cut downregulation in CAF1-p105/p180-deficient follicle cells at the mitotic stage. (A–A‴) Knockdown of Notch restored Cut expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in A) at the mitotic stage. Cut staining is shown in A′. (B–B‴) Knockdown of Notch abolished the induction of E(spl)mβ-CD2 expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in B) at the mitotic stage. CD2 staining is shown in B′. (C–C‴) Knockdown of Notch restored Eya expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in C) at the mitotic stage. Eya staining is shown in C′. (D–D‴) Knockdown of Notch restored Cut expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in D) at the mitotic stage. Cut staining is shown in D′. (E–E‴) Knockdown of Notch abolished the induction of Hnt expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in E) at the mitotic stage. Hnt staining is shown in (E′). (F–F‴) Immunostaining of the Notch <t>intracellular</t> <t>domain</t> (NICD) was performed to examine the knockdown efficiency of the Notch RNAi in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in F). NICD staining is shown in F′. DNA staining with DAPI is shown in A‴,B‴,C‴,D‴,E‴,F‴. The genotypes of GFP-negative and GFP-positive cell clones are indicated in black and green boxes, respectively, on the left sides of image panels. Scale bars: 10 μm.
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    Notch is required for premature activation of Notch target expression and Cut downregulation in CAF1-p105/p180-deficient follicle cells at the mitotic stage. (A–A‴) Knockdown of Notch restored Cut expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in A) at the mitotic stage. Cut staining is shown in A′. (B–B‴) Knockdown of Notch abolished the induction of E(spl)mβ-CD2 expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in B) at the mitotic stage. CD2 staining is shown in B′. (C–C‴) Knockdown of Notch restored Eya expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in C) at the mitotic stage. Eya staining is shown in C′. (D–D‴) Knockdown of Notch restored Cut expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in D) at the mitotic stage. Cut staining is shown in D′. (E–E‴) Knockdown of Notch abolished the induction of Hnt expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in E) at the mitotic stage. Hnt staining is shown in (E′). (F–F‴) Immunostaining of the Notch <t>intracellular</t> <t>domain</t> (NICD) was performed to examine the knockdown efficiency of the Notch RNAi in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in F). NICD staining is shown in F′. DNA staining with DAPI is shown in A‴,B‴,C‴,D‴,E‴,F‴. The genotypes of GFP-negative and GFP-positive cell clones are indicated in black and green boxes, respectively, on the left sides of image panels. Scale bars: 10 μm.
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    Image Search Results


    Notch is required for premature activation of Notch target expression and Cut downregulation in CAF1-p105/p180-deficient follicle cells at the mitotic stage. (A–A‴) Knockdown of Notch restored Cut expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in A) at the mitotic stage. Cut staining is shown in A′. (B–B‴) Knockdown of Notch abolished the induction of E(spl)mβ-CD2 expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in B) at the mitotic stage. CD2 staining is shown in B′. (C–C‴) Knockdown of Notch restored Eya expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in C) at the mitotic stage. Eya staining is shown in C′. (D–D‴) Knockdown of Notch restored Cut expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in D) at the mitotic stage. Cut staining is shown in D′. (E–E‴) Knockdown of Notch abolished the induction of Hnt expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in E) at the mitotic stage. Hnt staining is shown in (E′). (F–F‴) Immunostaining of the Notch intracellular domain (NICD) was performed to examine the knockdown efficiency of the Notch RNAi in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in F). NICD staining is shown in F′. DNA staining with DAPI is shown in A‴,B‴,C‴,D‴,E‴,F‴. The genotypes of GFP-negative and GFP-positive cell clones are indicated in black and green boxes, respectively, on the left sides of image panels. Scale bars: 10 μm.

    Journal: Journal of Cell Science

    Article Title: Inhibition of Notch signaling by the p105 and p180 subunits of Drosophila chromatin assembly factor 1 is required for follicle cell proliferation

    doi: 10.1242/jcs.224170

    Figure Lengend Snippet: Notch is required for premature activation of Notch target expression and Cut downregulation in CAF1-p105/p180-deficient follicle cells at the mitotic stage. (A–A‴) Knockdown of Notch restored Cut expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in A) at the mitotic stage. Cut staining is shown in A′. (B–B‴) Knockdown of Notch abolished the induction of E(spl)mβ-CD2 expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in B) at the mitotic stage. CD2 staining is shown in B′. (C–C‴) Knockdown of Notch restored Eya expression in CAF1-p180 RNAi-expressing follicle cell clones (GFP-positive in C) at the mitotic stage. Eya staining is shown in C′. (D–D‴) Knockdown of Notch restored Cut expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in D) at the mitotic stage. Cut staining is shown in D′. (E–E‴) Knockdown of Notch abolished the induction of Hnt expression in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in E) at the mitotic stage. Hnt staining is shown in (E′). (F–F‴) Immunostaining of the Notch intracellular domain (NICD) was performed to examine the knockdown efficiency of the Notch RNAi in homozygous CAF1-p10536 MARCM mutant follicle cell clones (GFP-positive in F). NICD staining is shown in F′. DNA staining with DAPI is shown in A‴,B‴,C‴,D‴,E‴,F‴. The genotypes of GFP-negative and GFP-positive cell clones are indicated in black and green boxes, respectively, on the left sides of image panels. Scale bars: 10 μm.

    Article Snippet: The following antibodies were used: mouse anti-Cut, 1:15 (2B10); mouse anti-Eya, 1:10 (eya10H6); mouse anti-Hnt (1G9), 1:15 (1G9); mouse anti-N ICD , 1:50 (C17.9C6) (all from the Developmental Studies Hybridoma Bank; DSHB); rabbit anti-PH3, 1:200 (catalog no. 06-570, Millipore); rabbit anti-β-galactosidase, 1:5000 (catalog no. 08559761, MP Biomedicals) and mouse anti-CD2, 1:50 (catalog no. MCA154G, AbD Serotec).

    Techniques: Activation Assay, Expressing, Knockdown, Clone Assay, Staining, Mutagenesis, Immunostaining